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Arteriosclerosis, Thrombosis, and Vascular Biology (ATVB)

Publisher :

American Heart Association Inc

Scopus Profile
Peer reviewed only
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Open Access
  • Medicine
  • Cardiology
  • Cardiovascular Medicine
e-ISSN :

1524-4636

Issue Frequency :

Monthly

Impact Factor :

8.7

p-ISSN :

1079-5642

Est. Year :

1995

Mobile :

8002428721

Country :

United States

Language :

English

APC :

YES

Impact Factor Assignee :

Google Scholar

Email :

atvb@atvb.org

Journal Descriptions

Arteriosclerosis, Thrombosis, and Vascular Biology (ATVB) publishes laboratory-based, translational, clinical and population research that is related to cardiovascular, peripheral vascular and cerebrovascular biology and disease. Areas of focus include atherosclerosis, lipid biology, arterial stiffening, thrombosis and hemostasis, and vascular and valvular biology. ATVB encourages submissions that use state-of-the-art approaches to illuminate mechanisms of disease and uncover new therapies and biomarkers through experimentation in animal and cellular models and in humans. The journal values novel technological developments—such as in bioengineering and with ‘omics platforms. ATVB maintains the highest standards of methodological and statistical rigor, prioritizes engagement with the research community (readers, authors, AHA/ATVB members, and our Early Career Editorial Board mentees) and publishes original research as well as scientific statements, viewpoints, opinions and consortium papers. Through dissemination of the highest quality research, ATVB supports the overall mission of the American Heart Association: to promote longer and healthier lives.


Arteriosclerosis, Thrombosis, and Vascular Biology (ATVB) is :

International, Peer-Reviewed, Open Access, Refereed, Medicine, Cardiology, Cardiovascular Medicine , Online or Print, Monthly Journal

UGC Approved, ISSN Approved: P-ISSN - 1079-5642, E-ISSN - 1524-4636, Established in - 1995, Impact Factor - 8.7

Not Provide Crossref DOI

Indexed in Scopus, PubMed

Not indexed in WoS, DOAJ, UGC CARE

Indexing

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Google Scholar
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Publications of ATVB

  • dott image August, 2000

Blood Pressure, LDL Cholesterol, and Intima-Media Thickness : A Test of the “Response to Injury” Hypothesis of Atherosclerosis

Abstract—The “response to injury” hypothesis is a plausible model of the development of atherosclerosis supported by observations from animal models. The present study uses epidemiolog...

  • dott image January, 2004

Progression of Carotid Intima-Media Thickness and Plasma Antioxidants: The Los Angeles Atherosclerosis Study

Objective— Recent epidemiologic and animal model data suggest that oxygenated carotenoids are protective against early atherosclerosis. We assessed the association between atherosclerotic ...

  • dott image August, 2004

A Summary of Implications of Recent Clinical Trials for the National Cholesterol Education Program Adult Treatment Panel III Guidelines

The Adult Treatment Panel III (ATP III) of the National Cholesterol Education Program (NCEP) issued an evidence-based set of guidelines on cholesterol management in 2001. Since the publicati...

  • dott image January, 2003

Obesity and Systemic Oxidative Stress: Clinical Correlates of Oxidative Stress in The Framingham Study

Objective— To determine the clinical conditions associated with systemic oxidative stress in a community-based cohort. Information regarding cardiovascular risk factors associated with sys...

  • dott image May, 2010

Biomarkers of the Osteoprotegerin Pathway: Clinical Correlates, Subclinical Disease, Incident Cardiovascular Disease, and Mortality

Objective— Experimental evidence identified the osteoprotegerin (OPG)/receptor activator of nuclear factor–κB (RANK)/RANK ligand (RANKL) pathway as a candidate system modulating vascula...

  • dott image January, 2014

Interleukin 1 Receptor 1 and Interleukin 1β Regulate Megakaryocyte Maturation, Platelet Activation, and Transcript Profile During Inflammation in Mic...

Objective— Interleukin 1 Receptor 1 (IL1R1) and its ligand, IL1β, are upregulated in cardiovascular disease, obesity, and infection. Previously, we reported a higher level of IL1R1 trans...

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