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Journal Photo for Cancer Research
Peer reviewed only Open Access

Cancer Research (CR)

Publisher : American Association for Cancer Research Inc
Cancer Biology Cancer Immunology Cancer Landscapes
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e-ISSN 1538-7445
p-ISSN 0008-5472
Issue Frequency Bi-Monthly
Impact Factor 16.6
Est. Year 1916
Mobile 2154409300
DOI YES
Country United States
Language English
APC YES
Impact Factor Assignee Google Scholar
Email cancerres@aacr.org

Journal Descriptions

Cancer Research publishes impactful original studies, reviews, and opinion pieces of high significance to the broad cancer research community. Cancer Research seeks manuscripts that offer conceptual or technological advances leading to basic and translational insights into cancer biology. Manuscripts that focus on convergence science, the bridging of two or more distinct areas of cancer research, are of particular interest. The main scope of the journal is captured in its primary subsections: Cancer Biology Cancer Immunology Cancer Metabolism and Molecular Mechanisms Therapeutic Development and Chemical Biology Translational Cancer Biology Cancer Landscapes Computational Cancer Biology and Technology Convergence Science Manuscripts containing findings that significantly move the field of cancer research forward. A potentially generalizable, broadly impactful conclusion, or an incisive rationale that could interest a broad audience. Results that have not been previously published in another tumor, cell line, or organoid model. Studies that include a clearly written title and abstract that communicate the study's impact to non-experts. Development or demonstration of a technological method that will greatly facilitate one or more areas of cancer research; such studies must demonstrate the potential of the approach by providing new biological insights to be considered. Studies with a high level of rigor and reproducibility of the methodologic approach at the time of submission: Multiple models Inclusion of age/sex as a variable Orthogonal approaches to support a hypothesis Proper controls Current techniques

Cancer Research (CR) is :-

  • International, Peer-Reviewed, Open Access, Refereed, Cancer Biology, Cancer Immunology, Cancer Landscapes, Convergence Science, Cancer Research, Cancer , Online or Print , Bi-Monthly Journal

  • UGC Approved, ISSN Approved: P-ISSN P-ISSN: 0008-5472, E-ISSN: 1538-7445, Established: 1916, Impact Factor: 16.6
  • Provides Crossref DOI

  • Indexed in: Scopus, WoS, PubMed

  • Not indexed in DOAJ, UGC CARE

Indexing

Publications of CR

Novel azetidine-based STAT3 inhibitors induce misfolded protein response, endoplasmic reticulum stress and mitophagy, and inhibit breast tumor growth ...
Nagendra Verma, June, 2022
Anticancer drugs that directly target Signal Transducer and Activator of Transcription (Stat)3 have remained elusive. We present azetidine-based small molecules as a new class of potent Stat...
Abstract 311: Diverse biological mechanisms drive resistance to Lorlatinib in ALK-rearranged Lung Cancer
Ahsan Z Rizvi July, 2019
Background: ALK rearrangements occur in 3-6% of patients (pts) with lung adenocarcinoma. Lorlatinib, is a novel third generation ALK tyrosine kinase inhibitor (TKI) with proven efficacy for ...
Participation of p53 Protein in the Cellular Response to DNA Damage
Bert Vogelstein December, 1991
The inhibition of replicative DNA synthesis that follows DNA damage may be critical for avoiding genetic lesions that could contribute to cellular transformation. Exposure of ML-1 myeloblast...
WAF1/CIP1 Is Induced in p53-mediated G1 Arrest and Apoptosis
Bert Vogelstein January, 1994
The tumor growth suppressor WAF1/CIP1 was recently shown to be induced by p53 and to be a potent inhibitor of cyclin-dependent kinases. In the present studies, we sought to determine the rel...
p21 Is Necessary for the p53-mediated G1 Arrest in Human Cancer Cells1
Bert Vogelstein November, 1955
DNA-damaging agents induce a p53-dependent G1 arrest that may be critical for p53-mediated tumor suppression. It has been suggested that p21WAF1/CIP1, a cdk inhibitory protein transcriptiona...
p53 Gene Mutations Occur in Combination with 17p Allelic Deletions as Late Events in Colorectal Tumorigenesis1
Bert Vogelstein December, 1990
Coordinate loss of one copy of the p53 gene and mutation of the remaining copy occur in colorectal carcinomas and in many other human malignancies. However, the prevalence of p53 gene mutati...
Mutational Analysis of the APC/β-Catenin/Tcf Pathway in Colorectal Cancer1
Bert Vogelstein March, 1998
Microsatellite Instability and Mutations of the Transforming Growth Factor β Type II Receptor Gene in Colorectal Cancer1
Bert Vogelstein December, 1995
The TGFβ type II receptor (RII) was found to be mutated within a polyadenine tract in 100 of 111 (90%) colorectal cancers with microsatellite instability. Other polyadenine tracts of simila...
Replication Errors in Benign and Malignant Tumors from Hereditary Nonpolyposis Colorectal Cancer Patients1
Bert Vogelstein April, 1994
A replication error (RER) phenotype has been documented both in sporadic colorectal tumors and in tumors from patients with hereditary nonpolyposis colorectal cancer (HNPCC). In the current ...
Topological Control of p21WAF1/CIP1 Expression in Normal and Neoplastic Tissues1
Bert Vogelstein July, 1995
The p53-regulated gene product p21WAF1/CIP1 is the prototype of a family of small proteins that negatively regulate the cell cycle. To learn more about p21WAF1/CIP1 regulation in vivo, monoc...