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Journal Photo for PLOS Genetics
Peer reviewed only Open Access

PLOS Genetics (PG)

Publisher : Public Library of Science
Disease biology Evolution Gene expression
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e-ISSN 1553-7404
p-ISSN 1553-7390
Issue Frequency Monthly
Est. Year 2005
Mobile 19172140095
Country United States
Language English
APC YES
Impact Factor Assignee Google Scholar
Email gbarsh@plos.org

Journal Descriptions

Genetics and genomics research has grown at a bewildering pace in the past 15 years. The techniques of these fields are being applied to a wealth of biological questions and experimental systems. PLOS Genetics reflects the full breadth and interdisciplinary nature of this research by publishing outstanding original contributions in all areas of biology. PLOS Genetics publishes human studies, as well as research on model organisms—from mice and flies, to plants and bacteria. Our emphasis is on studies of broad interest that provide significant insight into a biological process or processes. Topics include (but are not limited to) gene discovery and function, population genetics, genome projects, comparative and functional genomics, medical genetics, disease biology, evolution, gene expression, complex traits, chromosome biology, and epigenetics

PLOS Genetics (PG) is :-

  • International, Peer-Reviewed, Open Access, Refereed, Disease biology, Evolution, Gene expression , Online or Print , Monthly Journal

  • UGC Approved, ISSN Approved: P-ISSN P-ISSN: 1553-7390, E-ISSN: 1553-7404, Established: 2005,
  • Does Not Provide Crossref DOI

  • Indexed in: Scopus, DOAJ

  • Not indexed in WoS, PubMed, UGC CARE

Indexing

Publications of PG

Genome-Wide Association Study of Coronary Heart Disease and Its Risk Factors in 8,090 African Americans: The NHLBI CARe Project
Emelia J. Benjamin February, 2011
Coronary heart disease (CHD) is the leading cause of mortality in African Americans. To identify common genetic polymorphisms associated with CHD and its risk factors (LDL- and HDL-cholester...
Loss of ATRX, Genome Instability, and an Altered DNA Damage Response Are Hallmarks of the Alternative Lengthening of Telomeres Pathway
Bert Vogelstein July, 2012
The Alternative Lengthening of Telomeres (ALT) pathway is a telomerase-independent pathway for telomere maintenance that is active in a significant subset of human cancers and in vitro immor...