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Antioxidants and Redox Signaling (ARS)

Publisher :

Mary Ann Liebert Inc

Scopus Profile
Peer reviewed only
Scopus Profile
Open Access
  • Medicine
  • Biochemistry
  • Molecular Biology

    • +1

e-ISSN :

1557-7716

Issue Frequency :

Continuously

Impact Factor :

6.6

p-ISSN :

1523-0864

Est. Year :

1999

Mobile :

9147402100

Country :

United States

Language :

English

APC :

YES

Impact Factor Assignee :

Google Scholar

Email :

cksen@iu.edu

Journal Descriptions

Antioxidants & Redox Signaling (ARS) is the leading peer-reviewed journal dedicated to understanding the vital impact of oxygen and oxidation-reduction (redox) processes on human health and disease. The Journal explores key issues in genetic, pharmaceutical, and nutritional redox-based therapeutics. Cutting-edge research focuses on structural biology, stem cells, regenerative medicine, epigenetics, imaging, clinical outcomes, and preventive and therapeutic nutrition, among other areas. ARS has expanded to create two unique foci within one journal: ARS Discoveries and ARS Therapeutics. ARS Discoveries (24 issues) publishes the highest-caliber breakthroughs in basic and applied research. ARS Therapeutics (12 issues) is the first publication of its kind that will help enhance the entire field of redox biology by showcasing the potential of redox sciences to change health outcomes.


Antioxidants and Redox Signaling (ARS) is :

International, Peer-Reviewed, Open Access, Refereed, Medicine, Biochemistry, Molecular Biology, Cell Biology , Online or Print, Continuously Journal

UGC Approved, ISSN Approved: P-ISSN - 1523-0864, E-ISSN - 1557-7716, Established in - 1999, Impact Factor - 6.6

Not Provide Crossref DOI

Indexed in Scopus, PubMed

Not indexed in WoS, DOAJ, UGC CARE

Indexing

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Publications of ARS

Original Article
  • dott image November, 2022

Interleukin-33 Induces Neutrophil Extracellular Trap (NET) Formation and Macrophage Necroptosis via Enhancing Oxidative Stress and Secretion of Proath...

Interleukin-33 (IL-33) acts as an 'alarmin', and its role has been demonstrated in driving immune regulation and inflammation in many human diseases. However, the precise mechanism of action...

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