Subhadip Raychaudhuri
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About
Subhadip Raychaudhuri did his doctoral studies in Physics at University of Rochester (2002). Much of his PhD work was at the interface of non-equilibrium statistical physics, chemical engineering/chemistry and computational simulation. Subhadip received his postdoctoral training in the chemical engineering department at University of California Berkeley (2001-04). His postdoctoral research focused on computational biology and biophysics of T lymphocyte activation. Subhadip worked as an assistant professor at University of California Davis (Biomedical Engineering, 2004 – 2012), and, as an associate professor at Indraprastha Institute of Information Technology Delhi (Computational Biology, 2013-18). Subhadip also held a visiting professor position at Indian Association for the Cultivation of Science, Kolkata. Much of his recent interests are focused on solving biological problems utilizing interdisciplinary approaches and computation. In 2008, Subhadip received a R01 grant from the National Institute of Health USA (approx. USD 1M in funding) to develop kinetic Monte Carlo based computational simulation models of B lymphocyte activation pathways. Subhadip has more than 10 years of experience teaching undergraduate (UG) and PhD students. He has expertise in teaching science and technology courses and taught computational biology, biophysics, mathematical biology, physics courses.
Skills & Expertise
Biophysics
Quantum Mechanics
Research Interests
Biophysics
Mathematical Biology
Classical Physics
Quantum Mechanics
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Experience
Associate Professor
Associate Professor
- Associate Professor, Computational Biology, Indraprastha Institute of Information Technology, Delhi
Assistant Professor
- Assistant Professor, Biomedical Engineering, University of California Davis
Postdoctoral Researcher
- Postdoctoral Researcher, Chemical Engineering, University of California Berkeley
Education
University of Rochester
University of Calcutta
Presidency College
Publications (5)
Cancer cells frequently evade immune attack possibly by utilizing slow stochastic signaling through the mitochondrial cell death pathway and thus keeping apoptotic activation immunologically silent. A...
Biomimetic pro-apoptotic agents (e.g., BH3 mimetics) have been shown to activate the intrinsic death pathway (Type 2 apoptosis) selectively in cancer cells, a mechanism that can be key to developing s...
Apoptotic cell death is coordinated through two distinct (type 1 and type 2) intracellular signaling pathways. How the type 1/type 2 choice is made remains a central problem in the biology of apoptosi...
Cancer chemoresistance (including adaptive resistance) has emerged as a barrier in developing successful chemotherapeutic strategies. We use Monte Carlo simulation based single cell analysis to provid...
Death ligand mediated apoptotic activation is a mode of cell death that is widely used in cellular and physiological situations. Interest in studying death ligand induced apoptosis has increased due t...
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