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Molecular Cell (MC)

Publisher :

Cell Press

Scopus Profile
Peer reviewed only
Scopus Profile
Open Access
  • Biochemistry
  • Molecular Biology
  • Cell Biology
e-ISSN :

1097-4164

Issue Frequency :

Semi-monthly

p-ISSN :

1097-2765

Est. Year :

1997

Mobile :

16173972800

Country :

United States

Language :

English

APC :

YES

Impact Factor Assignee :

Google Scholar

Email :

bplosky@cell.com

Role In Research Journal

Brian Plosky
Bert Vogelstein

Editorial Board Member

Journal Descriptions

Molecular Cell aims to publish the best in molecular biology and beyond. In addition to fundamental cellular processes, we are interested in papers that show how molecular biology can be used to go beyond basic mechanisms to discover new biology, understand diseases, and to create new tools. Topic areas of interest include (but are not necessarily limited to): The Central Dogma of Molecular Biology - relating to the transmission of information between DNA, RNA, and proteins Cellular homeostasis - relating to turnover of proteins and organelles, mechanisms of cell death, cell fate, signaling, innate immunity, metabolism, and infection Tools for studying the above processes including - omics, genome editing, structural biology, microscopy, as well as AI, machine learning, and other computational approaches Understanding how molecular mechanisms are rewired or go awry in contexts such as cancer, metabolic disease, neurodegeneration, and aging or in other contexts including plants and microbes


Molecular Cell (MC) is :

International, Peer-Reviewed, Open Access, Refereed, Biochemistry, Molecular Biology, Cell Biology , Online or Print, Semi-monthly Journal

UGC Approved, ISSN Approved: P-ISSN - 1097-2765, E-ISSN - 1097-4164, Established in - 1997, Impact Factor

Not Provide Crossref DOI

Indexed in Scopus

Not indexed in WoS, DOAJ, PubMed, UGC CARE

Indexing

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Scopus
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Elsevier
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Science Direct

+2 More

Publications of MC

Research Article
  • dott image December, 1997

14-3-3σIs a p53-Regulated Inhibitor of G2/M Progression

Exposure of colorectal cancer (CRC) cells to ionizing radiation results in a cell-cycle arrest in G1 and G2. The G1 arrest is due to p53-mediated induction of the cyclin-dependent kinase inh...

Research Article
  • dott image March, 2001

PUMA Induces the Rapid Apoptosis of Colorectal Cancer Cells

Through global profiling of genes that were expressed soon after p53 expression, we identified a novel gene termed PUMA (p53 upregulated modulator of apoptosis). The protein encoded by PUMA ...

Research Article
  • dott image March, 1998

Human Smad3 and Smad4 Are Sequence-Specific Transcription Activators

Mounting evidence indicates that Smad proteins are required for TGFβ signaling, but the way(s) in which Smad proteins propagate this signal is unclear. We found that two human Smad proteins...

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