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Journal Photo for Cancer Cell
Peer reviewed only Open Access

Cancer Cell (CC)

Publisher : Cell Press
Biochemistry Molecular Biology Cancer Research
Claim Your Journal Get PRC Visit Website
e-ISSN 1878-3686
p-ISSN 1535-6108
Issue Frequency Monthly
Est. Year 1996
Mobile 6173972800
Country United States
Language English
APC YES
Impact Factor Assignee Google Scholar
Email smao@cell.com

Journal Descriptions

Cancer Cell provides a high-profile forum to promote major advances in cancer research and oncology. The primary criterion for considering manuscripts is whether the studies provide major advances into answering important questions relevant to naturally occurring cancers. Cancer Cell particularly welcomes translational research. Cancer Cell is also interested in publishing clinical investigations, in particular those that lead to establishing new paradigms in the treatment, diagnosis, or prevention of cancers; those that provide important insights into cancer biology beyond what has been revealed by preclinical studies; and those that are mechanism-based proof-of-principle clinical studies.

Cancer Cell (CC) is :-

  • International, Peer-Reviewed, Open Access, Refereed, Biochemistry, Molecular Biology, Cancer Research, Oncology, Medicine, Cell Biology , Online or Print , Monthly Journal

  • UGC Approved, ISSN Approved: P-ISSN P-ISSN: 1535-6108, E-ISSN: 1878-3686, Established: 1996,
  • Does Not Provide Crossref DOI

  • Indexed in: Scopus

  • Not indexed in WoS, DOAJ, PubMed, UGC CARE

Indexing

Role In Research Journal

Bert Vogelstein Profile Photo
Bert Vogelstein
Editorial Advisor

Publications of CC

Mutant PIK3CA promotes cell growth and invasion of human cancer cells
Bert Vogelstein June, 2005
PIK3CA is mutated in diverse human cancers, but the functional effects of these mutations have not been defined. To evaluate the consequences of PIK3CA alterations, the two most common mutat...
Histone modifications and silencing prior to DNA methylation of a tumor suppressor gene
Bert Vogelstein January, 2003
We attempted to answer two central questions about epigenetic silencing of the tumor suppressor gene p16INK4a in this study: (1) whether the maintenance of associated histone modifications i...