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Cancer Cell (CC)

Publisher :

Cell Press

Scopus Profile
Peer reviewed only
Scopus Profile
Open Access
  • Biochemistry
  • Molecular Biology
  • Cancer Research

    • +3

e-ISSN :

1878-3686

Issue Frequency :

Monthly

p-ISSN :

1535-6108

Est. Year :

1996

Mobile :

6173972800

Country :

United States

Language :

English

APC :

YES

Impact Factor Assignee :

Google Scholar

Email :

smao@cell.com

Role In Research Journal

Steve Mao
Bert Vogelstein

Editorial Advisor

Journal Descriptions

Cancer Cell provides a high-profile forum to promote major advances in cancer research and oncology. The primary criterion for considering manuscripts is whether the studies provide major advances into answering important questions relevant to naturally occurring cancers. Cancer Cell particularly welcomes translational research. Cancer Cell is also interested in publishing clinical investigations, in particular those that lead to establishing new paradigms in the treatment, diagnosis, or prevention of cancers; those that provide important insights into cancer biology beyond what has been revealed by preclinical studies; and those that are mechanism-based proof-of-principle clinical studies.


Cancer Cell (CC) is :

International, Peer-Reviewed, Open Access, Refereed, Biochemistry, Molecular Biology, Cancer Research, Oncology, Medicine, Cell Biology , Online or Print, Monthly Journal

UGC Approved, ISSN Approved: P-ISSN - 1535-6108, E-ISSN - 1878-3686, Established in - 1996, Impact Factor

Not Provide Crossref DOI

Indexed in Scopus

Not indexed in WoS, DOAJ, PubMed, UGC CARE

Indexing

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Scopus
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Science Direct
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WorldCat

+3 More

Publications of CC

Research Article
  • dott image June, 2005

Mutant PIK3CA promotes cell growth and invasion of human cancer cells

PIK3CA is mutated in diverse human cancers, but the functional effects of these mutations have not been defined. To evaluate the consequences of PIK3CA alterations, the two most common mutat...

Research Article
  • dott image January, 2003

Histone modifications and silencing prior to DNA methylation of a tumor suppressor gene

We attempted to answer two central questions about epigenetic silencing of the tumor suppressor gene p16INK4a in this study: (1) whether the maintenance of associated histone modifications i...

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